Lung tissue from normoxia- or hyperoxia-treated control or SCNN1B overexpression mice were processed with a redox proteomics workflow and analyzed by LC-MS/MS with TMT based quantification. Data was searched with MS-GF+ using PNNL's DMS Processing pipeline.
[doi:10.25345/C55D8NJ9M]
[dataset license: CC0 1.0 Universal (CC0 1.0)]
Keywords: redox proteomics ; hyperoxia ; lung ; S-glutathionylation ; thiol oxidation ; SCNN1B
Principal Investigators: (in alphabetical order) |
Wei-Jun Qian, Pacific Northwest National Laboratory, United States |
Submitting User: | alchemistmatt |
Zhang T, Day NJ, Gaffrey M, Weitz KK, Attah K, Mimche PN, Paine R, Qian WJ, Helms MN.
Regulation of hyperoxia-induced neonatal lung injury via post-translational cysteine redox modifications.
Redox Biol. Epub 2022 Jul 19.
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